War against a joker

 

The pharmaceutical industry and its virologists are currently trying, for transparent reasons, to define the pathogen SARS-CoV2 as a stable hostile entity. For the "war against the virus", the danger is needed, represented by a spiked ball. A "corona-free world" is the declared goal of vaccine-obsessed Bill Gates and his political friends. Also, with regard to a possible vaccination, they try to give us the illusion of a clearly definable enemy in the world of viruses. After all, this is the prerequisite for the testing business and the governmental enforcement of a worldwide vaccination policy that is risk-free for vaccine manufacturers. Today, on 4.5.2020, an online donor conference on the "Creation of a corona-free world" (ARD-Tagesschau) is taking place and Chancellor Merkel promises to spend billions of our tax money on it. From a scientific point of view, all these efforts are - to put it mildly - dangerous aberrations. I am not yet talking about the profiteers of this madness.

 

Life is not trivial and calculable

It is absolutely certain that the SARS virus is also constantly and rapidly changing. And what good is a vaccination against something that has long since changed incalculably? Our immune system also reacts unpredictably. Cross-immunities? Immune memory? The specificity and significance of tests is quickly fading. So does the effect of a vaccine.

That's why proof of immunity is a farce, and if it becomes law, a health hazard that cannot be justified. For the same reason, mass vaccinations against respiratory viruses are a risky nonsense and may cause bodily injury. With rapidly changing pathogens, as with influenza vaccination, success is a matter of luck. Only afterwards can it be determined whether the vaccinated persons were better off than the non-vaccinated. This remains a good deal, since an evidence-based prior benefit assessment will of course never be possible. Up to now, other viruses have been happy and spread where one type of virus has become difficulties by vaccination.

 

The Wuhan viruses are long gone

It's all about the extremely high mutation rate of RNA viruses, which includes SARS-CoV2. The rate is impressively demonstrated by the website CoV-Glue, " Amino acid analysis for the SARS-CoV-2 pandemic", which shows that for SARS-CoV2 the mutation rate is very high in a very short period of about 4 months.

· 7237 non-synonymous, i.e. amino acid changing mutations (replacements),

· 6 insertions (inserting additional bases) and

· 87 deletions (loss of bases in the gene sequence) in found gene sequences. .

That is for a genome that itself consists of only about 30,000 bases an enormous number of mutations, insertions and deletions in a very short time. And these are only the data of a few thousand SARS-CoV2 viruses that have been sequenced. Nature knows many, many more. Non-synonymous mutations cause other amino acids to be inserted into the virus' proteins. These thus change the chemical properties of these proteins. These mutations accumulate within weeks(!), as the data show. Insertions and deletions are of special importance, because they can lead to a frame shift, where the whole subsequent chain is read differently. There are also synonymous mutations which, although they do not change the primary structure of the SARS-CoV2 proteins, can still play a role in diagnostics. In addition, there are still many open questions about further effects of synonymous mutations. Doubts about the PCR test, existed from the beginning: Li et al, "Stability Issues of RT-PCR Testing of SARS-CoV-2 for Hospitalized Patients Clinically Diagnosed with COVID-19", J Med Virol. 2020 Mar 26. doi: 10.1002/jmv.25786,

 

Snapshots of a neverending story

Another important point is that the SARS-CoV2 viruses sequenced so far show an extremely small section of nature. In relation to the total genome of all corona viruses in all humans, this section is of little importance. It must also be remembered that there are not only SARS-CoV2, but also other human coronaviruses - and they also mutate. Otherwise one would not have ended up with the SARS-CoV2 virus. Computer analyses, e.g. of the family tree of SARS-CoV2 (phylogenetic analysis), are very problematic based on this extremely small section of nature as it exists today. For the last 15 years, corona viruses have hardly been considered, neither in humans nor in animals. The vast majority of the coronavirus gene sequences in the databases originate from the last 4 months or are 15 years old. The criticism of this is not new, especially the very inhomogeneous geographical distribution of the gene sequences found, see Mavian et al, "Regaining perspective on SARS-CoV-2 molecular tracing and its implications", medrxiv, 20.3. 2020, : "However, in a new tree inferred just one week later, when more than 135 new full genome sequences were made available on GISAID (Figure S2), the direct link between Germany and Italy has disappeared due to the additional clustering of previously unsampled sequences from Portugal, Brazil, Wales and Netherland (Figure 2b).

 

Zoonosis?  Man is also a zoon!

Some publications say that haplotypes (i.e. genetic patterns) of SARS-CoV2 have already disappeared again, i.e. are no longer found in newly sequenced base sequences. SARS(1) has also disappeared again for a long time. What sense does a genetic distance between two gene sequences make then?

But virology is doing what it has been doing for the last 30 years. It finds a viral gene sequence that it did not yet know and declares the newly discovered virus a death virus. For this to work, it needs the zoonosis hypothesis.

Without it, it doesn't work. Because only thanks to this hypothesis that a pathogen has been newly transferred from an animal host to humans in Wuhan at the fish market at the end of 2019, can a general assumption be made that the pathogen has an increased pathogenicity (= an increased potential to cause illness). The pathogen is new to humans, which is why humans, in contrast to the original hosts, have not had the opportunity to adapt to the new pathogen.

This approach works so well because it is always assumed that patients are seriously ill, who then usually suffer from several diseases (multimorbid), and people with no or weak symptoms are not tested. And immediately you have the apparent proof that positive testing and death go hand in hand.

As proof of this, Drosten, Wieler & Co. hold up two gene sequences, one from an animal corona virus and one from a human corona virus, and point to the large genetic distance. This assertion can be made so easily because there is no data for anything in between. Nobody has measured animal and human coronaviruses in the necessary density in the last 15 years. A very large number of SARS-CoV2 positive people show no or only mild symptoms. The proportion of asymptomatic people who do not show symptoms is said to be 50 - 70%. This is not possible without the immune system having been prepared for the virus. This suggests a continuous development up to SARS-CoV2 (and its many variants).

 

 

Talking about origin only shows where you started thinking

Increasingly, phylogenetic analyses are also being carried out in an attempt to construct a different family tree from the numerous different gene sequences; contrary to the hypothesis that SARS-CoV2 was newly created by zoonosis at the end of 2019, this is not the case. Analyses of this kind are problematic due to the very short sample period. However, this is all that is currently available. In addition, the zero point, i.e. the reference sequence against which mutations are evaluated, is arbitrarily chosen. Only in China has the new PCR test been started and the first gene sequences have been generated. Only then did they do the same in other parts of the world. What is a forward and backward mutation is a relative statement in relation to the reference sequence. The supposed spread only reflects the geographical sequence of the tests. Nevertheless, the alarmists at Johns Hopkins continue to use this elsewhere.

 

 

We should finally give up the image of a "family tree" - the image of recursive networks appears more appropriate

The diversity of SARS-CoV2 is so great that clusters are formed even in small samples. It is no longer possible to clearly assign how the virus got to New York. Cf. Gonzalez-Reiche et al., "Introductions and early spread of SARS-CoV-2 in the New York City area", April 16, 2020, medxriv: "Phylogenetic analysis of 84 distinct SARS-CoV2 genomes indicates multiple, independent but isolated introductions mainly from Europe and other parts of the United States. Moreover, we find evidence for community transmission of SARS-CoV2 as suggested by clusters of related viruses found in patients living in different neighborhoods of the city." These clusters must have formed in the USA in the last 3 months. It is the SARS-CoV2 and everything that led to SARS-CoV2 is much older.

Alternative interpretations of the same data(!) from the gene databases suggest that SARS-CoV2 has been spreading in humans for 40+ years. See Chaw et al, "The origin and underlying driving forces of the SARS-CoV-2 outbreak", April 14, 2020, bioxriv:

“We have to point out that the TMRCA estimation is strongly influenced by the genome sampling scheme. Since the earliest available genome was sampled on 12/24/2019 almost one month after the outbreak, the real origin of the current outbreak may actually be earlier than our estimation.”

“Assuming a synonymous substitution rate of 2.9x10-3/site/year, the recombination was estimated to have occurred approximately 40 years ago (95% HPD : 31-69 years; divergence time (t) = divergence (dS)/(substitution rate x 2 x 3), considering dS in RBD is 3-fold of genome average). The amino acids in the RBD region of the two genomes have been maintained by natural selection ever since, while synonymous substitutions have been accumulated. If this is true, SARS-CoV-2 may have circulated cryptically among humans for years before being recently noticed.”

These are very recent evaluations and it remains to be seen whether this preprint will make it through the much-vaunted peer review process by which science "regulates" itself and the state of knowledge.

 

What glyphosate is to fields, antivirals are to humans

Viruses are probably as old as bacteria. The vertebrate immune system is the only one capable of harmonizing the body with the ever-new virus variants. This is exactly what it was created for in the course of evolution. The group of vertebrates originated about 500 million years ago and the concept of the immune system is possibly even older. 500 million years ago, the vertebrate immune system, at the preliminary endpoint of which is man, developed in close contact with the viruses. However, modern medicine uses corticosteroids to suppress the immune response, as shown by many COVID-19 treatment protocols, particularly from the USA.

In the meantime, the industry is constantly advertising new drugs. Where are they used? 98% of test positives show no or only mild symptoms. But the media uncritically mix test and infection, infection and symptomatic disease, symptomatic disease and death. The severely affected persons are 80+ on average and in the vast majority of cases suffer from severe pre-existing conditions. Nobody can say how many of them have died as a result of premature ventilation. In the meantime, intensive care patients are still being weaned off breathing with fentanyl, among other things, otherwise artificial respiration will not work. These patients wake up, if at all, as junkies. Katherine Ellen Foley, "Some drugs used to keep coronavirus patients on ventilators are in short supply", Quartz, March 31, 2020,

 

The fear is coming out of the ICU, but what's going on?

The ICU's showing what it can do. In addition, there are drugs like Remdesivir, which has been too toxic for the Ebola therapy and fails in further tests. Josh Farkas. "PulmCrit – Eleven reasons the NEJM paper on remdesivir reveals nothing", April 11, 2020, All doubts have not prevented Remdesivir from being released for use in the USA by the flash method. Or chloroquine and azithromycin combination therapies, which can lead to cardiac arrest in people with previous cardiovascular diseases. Or chloroquine monotherapy, which causes damage to red blood cells in people with glucose-6-phosphate dehydrogenase deficiency. Glucose-6-phosphate dehydrogenase deficiency is the most common hereditary genetic defect worldwide, with a frequency of up to 20-30% in Africa. Countries with a high proportion of African-born people are also particularly affected.

 

Wrong priorities and deceptive figures

Everything is initiated by a dilapidated test, hasty measures and a blind trust in  some nano part of the evolutionary machine nature, which is digitized in the gene databases and not justified by anything. Science and the politicians beleaving its advises will have to continue to pursue their self-made crisis. Although it is very doubtful that a virus that causes no(!) symptoms in 50 - 70% of infected people had arrived in Europe at the exact time the tests were started, as some now want to make us believe with the help of the curves for excess mortality. And this in a patient population in which 40% of intensive care patients came directly from the nursing home to the ICU in need of the most intensive care. See Matthias Thöns, "Very wrong priorities set and all ethical principles violated", Deutschlandfunk, 11.4.2020, "And we have to remember that the seriously ill COVID-19 patients, as the disease is called, are mostly very old people, many of whom are ill, 40 percent of whom come from nursing homes in need of the highest degree of care, and in Italy, out of 2,003 deaths, only three patients have been without serious pre-existing conditions. So it is a group that has usually and until now always received more palliative care than intensive care, and now a new disease is diagnosed and all these patients become intensive care patients."  In some countries there is an extra compensation for hospitals, if the DRG is upgraded to covid-19. This gets still more attractive, if the patient gets intubated. Medicare in US pays 20% more for DRGs with covid-19.

 

False alarms - a social autoimmune disease

In the current media climate and under immense political pressure to justify the draconian measures and their consequences, even retrospectively, it is very doubtful that there will be an independent scientific discussion on the zoonosis hypothesis of prudent risk assessment. For which research applications will money be granted? Evolution has been going on for millions of years, and in the last 20 years virologists have been conjuring up new death viruses every 3-5 years to threaten humans? MERS, SARS(1), SARS(2), swine flu, bird flu? This has gone really stupid.